If you log out, you will be required to enter your username and password the next time you visit. Reactive gastropathy is a condition that develops when substances such as non-steroidal anti-inflammatory drugs (NSAIDs), alcohol, or bile damage the cells that cover the inside of the stomach. Rights Reserved. Copyright 2023. 1983 Nov 12. Upper gi, antral mucosa w/ regenerative epithelial changes and vascular congestion. Am J Gastroenterol. Acute gastritis can persist anywhere from 2 to 10 days. Antral mucosa exhibiting the features of reactive gastropathy, including 'corkscrew-like' foveolar hyperplasia, a mucin depleted epithelium and bundles of hyperplastic smooth muscle arranged. Postoperative reflux gastritis: pathophysiology and long-term outcome after Roux-en-Y diversion. 1999 Jun 17. Etymologically, the. Special stains for H pylori are negative. Representative histology of healthy antral mucosa (A), antral . 303(3):136-8. Persistent epithelial damage may result in the release of platelet-derived growth factor (PDGF), which stimulates smooth muscle proliferation, followed by fibroblastic proliferation 12. This question is best answered by your gastroenterologist who had a good look at your tissue and pathology report. 2003 Apr. Stool changes. Genta RM. Antonia R Sepulveda, MD, PhD is a member of the following medical societies: American Association for Cancer Research, American Gastroenterological Association, American Society for Investigative Pathology, College of American Pathologists, United States and Canadian Academy of PathologyDisclosure: Nothing to disclose. https://www.askapollo.com/doctors/medical-gastroenterologist/hyderabad/dr-naveen-polavarapu. benign gastric antral mucosa with mild chronoc inactive gastitis focal intestinal metaplasia present? 16(4):325-41. The following are examples of well-known foods to eat and foods to avoid in case of gastritis: Related:Bowel movements: How often should you poop? N Engl J Med. Two other features, (4) acute and (5) chronic inflammatory cells, were graded separately; scores for each ranged from 0 (severe increase) to 3 (absence of inflammatory cells), for a combined score of 0 to 6. 352(25):2576-8. Mine said something similar. Gastric antral vascular ectasia (GAVE), also described as watermelon stomach, is characterized by rows or stripes of ectatic mucosal blood vessels that emanate from the pylorus and extend proximally into the antrum (Fig. The management of this condition may include discontinuance of NSAID use along with administration of proton pump inhibitors (PPIs) and prostaglandin analogues. 1999 Oct. 13(10):1273-85. We herein, through this case, would like to highlight that high clinical suspicion is required to diagnose . Aliment Pharmacol Ther. Gastroenterol Clin North Am. The presence of fibrin thrombi in the mucosal capillaries is a characteristic feature of GAVE rather than of reactive gastropathy. Although a correlation between histological evidence of chemical gastropathy and clinical manifestations, particularly risk of bleeding, is yet to be documented, reporting the suspicion of drug-induced gastric damage may help clinicians to identify patients that might benefit from change, reduction, or discontinuation of certain medications 5. Rom J Morphol Embryol. Tiredness and weakness can result in Foveolar Hyperplasia. Gastritis is usually not serious and can be treated through various treatment options. [11, 12] Prostaglandins are derived from arachidonic acid via the cyclooxygenase (COX) pathway. [23] Although the findings are not specific, several authors claim that weight loss and a hypochromic microcytic anemia are also associated features. [13], Although it is known that NSAIDs that selectively inhibit COX-2 cause significantly fewer GI complications than nonselective COX inhibitors do, it is still unclear whether administration of selective inhibitors results in less severe reactive gastropathy (chemical gastropathy). 2007 Jan. 131(1):86-90. J Clin Pathol. Antral Gastritis: Gastritis refers to inflammation of the inner stomach lining. Disclaimer: MyPathologyReport.ca is a registered not-for-profit charity (769563271RR0001). Gastritis and gastropathy may be chronic, developing slowly and lasting a long time, or acute, developing suddenly and lasting a short time. When associated with bile reflux secondary to partial gastrectomy, the lesions develop near the surgical stoma 18, but the more proximal oxyntic mucosa may also be affected. Re-seal . Antral Gastritis: Gastritis refers to inflammation of the inner stomach lining. I was diagnosed with functional dyspepsia and told to try brain-gut meds (antidepressants). 1. Helicobacter pylori-induced chronic gastritis and assessing risks for gastric cancer. [26]. Patients who had undergone gastric surgery were found to have a statistically significant increase in reflux gastritis score, gastric pH, and gastric bile acid concentration. [10] The various bile acid species differ in their capacity to cause injury to the gastric mucosa. Extreme stomach cramps and pain Shortness of breath or light-headedness Fatigue Vomiting blood or vomit that looks like ground coffee Sometimes you will have mild bleeding in your stomach but may. Wolfe MM, Lichtenstein DR, Singh G. Gastrointestinal toxicity of nonsteroidal antiinflammatory drugs. Erosive gastritis is gastric mucosal erosion caused by damage to mucosal defenses. Drinking milk causes an increase in stomach acid production, which might aggravate gastritis symptoms. [QxMD MEDLINE Link]. scant or minimal inflammation, i.e. Gastritis can develop quickly (acute gastritis) or gradually over time (chronic gastritis). Mucosal changes in reactive gastropathy are readily appreciated by comparison with normal gastric mucosa (see the image below). Most of the esophagus is lined by squamous mucosa. Northeast Ohio 216.444.7000 Appointments & Locations What is gastric antral mucosa with reactive Gastropathy? 340(24):1888-99. [QxMD MEDLINE Link]. Histology of the postoperative stomach before and after diversion of bile. Poor digestion, stretching, and increment of the stomach takes place during Foveolar Hyperplasia. [QxMD MEDLINE Link]. [5, 7, 21, 22], The clinical features associated with reactive gastropathy are determined by its underlying cause. Antral gastritis can cause excessive discomfort in the gastrointestinal tract of those affected. [QxMD MEDLINE Link]. Further symptoms of antral gastritis are noted, such as: decreased appetite; frequent heartburn; eructation (with reduced acidity - rotten, with increased - acid); an unpleasant aftertaste in the mouth (with reflux gastritis - bitter); A whitish or grayish-yellow coating on the surface of the tongue; nausea; vomiting; bloating and flatulence; Hence, its advisable to avoid consuming milk when recovering from gastritis. American Society for Investigative Pathology, United States and Canadian Academy of Pathology. Its characteristic feature is presence of coiled and branching antral glands. [16] but the more proximal oxyntic mucosa may also be affected. none? N Engl J Med. J Clin Pathol. Hum Pathol. Chemical or reactive gastritis is caused by injury to the gastric mucosa resulting from reflux of bile and pancreatic secretions into the stomach, but it can also be caused by exogenous. This finding, along with its distinctive endoscopic appearance ("watermelon stomach"), aids in the differentiation of GAVE from other similar-appearing entities. This condition can be caused by the following factors: Gastritis, if left untreated, can lead to serious complications like: Anemia: H. pylori can cause stomach ulcers or gastritis that bleed, resulting in a drop in the red blood cell count leading to anemia. This is a descriptive diagnosis. Abha Goyal, MD is a member of the following medical societies: American Society for Clinical Pathology, American Society of Cytopathology, College of American Pathologists, United States and Canadian Academy of PathologyDisclosure: Nothing to disclose. This medication can be purchased over the counter and works very well. what does this really mean? 39(5):700-4. Chemical gastritis and Helicobacter pylori related gastritis in patients receiving non-steroidal anti-inflammatory drugs: comparison and correlation with peptic ulceration. In the gastric mucosa, metaplasia, dysplasia, cancer, and foveolar hyperplasia (FH), a feature of reactive gastritis or gastropathy, were sought in the antrum and fundus. May mimic a (true) gastritis symptomatically and visually in an endoscopic examination. Most people do not experience noticeable effects of intestinal metaplasia. gi doctor told me to check up in a year. Am J Surg Pathol. Reactive gastropathy is characterized, histologically, by 21: If long-term use of nonsteroidal anti-inflammatory drugs (NSAIDs) leads to reactive gastropathy, your doctor may recommend that you stop taking NSAIDs, take a lower dose, or take a different medicine for pain. Despite the occasional development of stump carcinomas in postgastrectomy stomachs, reactive gastropathy is not a major risk factor for the development of gastric carcinoma. Aliment Pharmacol Ther2012;36:736743 https://doi.org/10.1111/apt.12031. The signs and symptoms of gastritis include: Gnawing or burning ache or pain (indigestion) in your upper abdomen that may become either worse or better with eating. Gastropathy indicates there is something wrong there, typically inf Dr. Charles Cattano and another doctor agree. A patchy or complete loss of the membrane mucin MUC1 was observed in 67% of cases. The surface may appear villiform. [QxMD MEDLINE Link]. [QxMD MEDLINE Link]. They generate the mucus inside the stomach that's accountable for shielding the tummy from the gastric acids. Furthermore, NSAIDs, being weak organic acids, can freely diffuse into the gastric epithelium. Endoscopic evaluation of the effects of aspirin, buffered aspirin, and enteric-coated aspirin on gastric and duodenal mucosa. It occurs when there's an expansion of unwanted foveolar cells inside the stomach tissues or tissue layer. Joshua P Cantor, MD Staff Pathologist, Department of Pathology, Abington Memorial Hospital Other forms are nonerosive. The vast majority of cases of gastric ulcers (70% to 90% . The predominant mechanism of NSAID-induced gastric injury involves decreased synthesis of mucosal prostaglandins13. Triad rarely present; mild inflammation common. 2.75. Mucin expression in reactive gastropathy: an immunohistochemical analysis. This was the only thing on my colonoscopy/endoscopy report that I don't understand. Chronic alcohol consumption can irritate and damage the stomach lining. 1991 Feb. 71(1):33-44. Gastritis and carditis. These alterations in mucin expression were shown in cases of both nonsteroidal anti-inflammatory drug (NSAID)-associated and bile refluxassociated reactive gastropathy (chemical gastropathy). The statements herein have not been evaluated by the Foods and Drugs Administration or Health Canada. It may range from mild to severe, triggering vomiting in some cases. This lining shields the stomach from the corrosive stomach acid that aids digestion by breaking down food. Gastritis is usually not serious and can be treated through various treatment options. Carrasco G, Corvalan AH. After a meal, avoid lying down for up to 2 to 3 hours. 1999 Oct. 13(10):1273-85. Everything you need to know, Feeling full or bloated during or after a meal. lack of large numbers of neutrophils and plasma . The following are common treatments for managing and treating antral gastritis: Antral gastritis, or gastritis in general, can be prevented by following healthy lifestyle habits and a balanced diet. [2, 3, 4] It has since become apparent, however, that the constellation of histologic features seen in reactive gastropathy is a nonspecific response to chemical injury of the gastric mucosa. Gastritis can develop quickly (acute gastritis) or gradually over time (chronic gastritis). A: Sections from the gastric antrum show portions of antral and oxyntic-type gastric mucosa with edema, vascular congestion and patchy chronic inflammation of the lamina propria. Reactive gastropathy is associated with inflammatory conditions throughout the gastrointestinal tract. Bile reflux is thought to play a key role in the development of dysplasia and carcinoma in the gastric remnant22. Severe reactive gastropathy with mucosal erosion of gastric antral mucosa (original magnification 200). Methods: We retrieved all patients younger than 18 years who had upper GI endoscopy with a pathology diagnosis of antral IM between 2009 and 2020. Histopathology. Mar 16, 2018 #6 46(4):269-74. This subreddit is designed for discussing gastritis and diseases related to gastritis, such as peptic (stomach) and duodenal ulcers. Press J to jump to the feed. Gastritis is a condition that leads to stomach lining inflammation, causing stomach pain, bloating, and nausea. 27:1348-54. The common underlying causes of reactive gastropathy include chronic bile reflux and long-term intake of nonsteroidal anti-inflammatory drugs (NSAIDs) 2. 20021610541-overviewDiseases & Conditions, encoded search term (Pathology of Reactive Gastropathy) and Pathology of Reactive Gastropathy, Helicobacter Pylori-Associated Active Gastritis, Fast Five Quiz: Helicobacter pyloriAssociated Gastritis, A Guide to Managing Gastric Intestinal Metaplasia, Prevalence and Prognostic Significance of Vitamin C Deficiency in Patients With Acute Upper Gastrointestinal Bleeding, AGA Offers Key Guidance on Managing Subepithelial GI Lesions, Early Satiety, Nausea, and Vomiting After Meals: Case Presentation. - Scant or minimal inflammatory cells (white arrow), i.e. Waxman HA. Characterized by scars and lesions, this form is considered difficult to treat. [Full Text]. The antrum is that portion of the lower stomach above the pylorus. 1993 Oct. 23(4):341-8. Differential diagnosis of reactive gastropathy. This can include everything from ulcers to gastritis. Privacy Policy. International Workshop on the Histopathology of Gastritis, Houston 1994. The term chemical gastropathy was recommended by the Updated Sydney System 7, because it indicates an underlying chemical injury that is not associated with infection 8. 2005. Only after we are satisfied with the products. 287(6403):1410-2. Genta, RM. Causes include bile reflux, non-steroidal anti-inflammatory drugs, alcohol, and smoking. Scores of 10 or higher were found to be highly characteristic of reactive gastropathy (chemical gastropathy). what that mean? Although decreasing in overall incidence with the routine use of histamine-2 (H 2 ) blockers, the overall death rate from peptic ulcer disease has remained stable. The diagnosis of reactive gastritis is made after a pathologist examines a tissue sample from the inside of the stomach under the microscope. [14] However, most of these COX-2 inhibitors have been withdrawn from the market or have had their indications drastically limited in view of their potential serious cardiovascular side effects. Maguilnik I, Neumann WL, Sonnenberg A, Genta RM. using current production and quality control standards. Having chronic inflammation with extensive intestinal metaplasia is the precursor for developing gastric cancer. The common underlying causes of reactive gastropathy include chronic bile reflux and long-term intake of nonsteroidal anti-inflammatory drugs (NSAIDs). 2. Maguilnik, I.; Neumann, WL. [QxMD MEDLINE Link]. The endoscopic findings of reactive gastropathy are mostly nonspecific. Ritchie WP Jr. Alkaline reflux gastritis. N Engl J Med. [1] because it indicates an underlying chemical injury that is not associated with infection. 1995 Apr. What is the Difference Between Piles, Fissures and Fistula? Another name for this condition is chemical gastropathy. Haber MM, Lopez I. Gastric histologic findings in patients with nonsteroidal anti-inflammatory drug-associated gastric ulcer. The antrum refers to the lower portion of the stomach. Diagram of the alkaline mucous layer in the stomach with mucosal defense mechanisms. An antral ulcer is a form of peptic ulcer as it is concentrated in the stomach lining. Are there neutrophils in the mucosa? The mucosal changes seen in reactive gastropathy are usually most prominent in the antrum and prepyloric region. I. Maguilnik W. L. Neumann A Sonnenberg R. M. Genta. Histopathology. As inflammation develops in the stomachs antrum, the digestion process and also the process of the food getting emptied into the intestine will get affected. H. pylori bacteria are the most common cause of persistent gastritis and peptic ulcer disease (stomach ulcers). 1996. Antral gastritis can cause excessive discomfort in the gastrointestinal tract of those affected. Am J Surg Pathol 1996; 20: 116181. If long-term use of nonsteroidal anti-inflammatory drugs (NSAIDs) leads to reactive gastropathy, your doctor may recommend that you stop taking NSAIDs, take a lower dose, or take a different medicine for pain. (Dec 2014). In humans, it is about 1 mm thick, and its surface is smooth, soft, and velvety. These gastric mucosal protection and ulcer healing effects are associated with antioxidant effects and the augmentation of prostaglandin E2 and suggest that P. cuspidatum might be a promising . [QxMD MEDLINE Link]. 1992 Feb. 45(2):135-9. 23(2):281-94. after finishing therapy. [QxMD MEDLINE Link]. [5] The term "chemical gastropathy" was recommended by the Updated Sydney System, (Nov 2005). [25]. J Clin Pathol. Mamoun Younes, MD is a member of the following medical societies: American College of Gastroenterology, American Gastroenterological Association, American Society of Clinical Oncology, College of American Pathologists, United States and Canadian Academy of PathologyDisclosure: Nothing to disclose. (black arrow), as a tortuosity in the "neck" region of the gastric glands. The updated Sydney System. Another name for this condition is chemical gastropathy. Histological features do not define NSAID-induced gastritis. Most people have a problem with their stomach at one time or another. 1985 Aug. 103(2):178-83. . This website also contains material copyrighted by 3rd parties. Seminars in Diagnostic Pathology Volume 22, Issue 4, November 2005, Pages 273-283. Wolf, EM. [QxMD MEDLINE Link]. What are reactive changes? Differential diagnosis of reactive gastropathy. [QxMD MEDLINE Link]. 1999 Jun 17. Malagelada JR, Phillips SF, Shorter RG, et al. The features are those of a chronic gastritis. Endoscopy pathology report. The densities of inflammatory cells and reactive atypia were scored at squamous, cardiac and oxyntocardiac mucosa of SCJ, antrum and body. Iijima K, Iwabuchi T, Ara N, et al. Histological features do not define NSAID-induced gastritis. The bile reflux may be visible 20. All Rights Reserved. Weir, MR.; Espaillat, R. (Jun 2015). Occasionally, focal intestinal metaplasia may be present. Reactive increase in gastric mucus secretion is an adaptive defense mechanism against low-dose aspirin-induced gastropathy. Reactive gastropathy is a relatively common finding in gastric biopsies; in most instances it is associated with either reflux of duodenal contents or therapy with nonsteroidal. Reactive increase in gastric mucus secretion is an adaptive defense mechanism against low-dose aspirin-induced gastropathy. Gastric erosive changes are a common cause of UGI bleeding, and hemorrhagic erosive gastritis is thought to be responsible for 10% to 20% of upper tract bleeding. . Am J Surg Pathol. In one study the risk of metaplasia > gastric cancer was 1 IN 39 PATIENTS OVER A 20 YEAR PERIOD. Reactive gastropathy, abbreviated RG, a relatively common pathology of the stomach. Medical attention is needed right away if this is observed. Burden WR, Hodges RP, Hsu M, O'Leary JP. Does anyone know what it means? N Engl J Med. Vere CC, Cazacu S, Comanescu V, Mogoanta L, Rogoveanu I, Ciurea T. Endoscopical and histological features in bile reflux gastritis. The accompanying histamine-mediated vascular response leads to edema and hyperemia. Watt PC, Sloan JM, Spencer A, Kennedy TL. Arrow points to smooth muscle fibers in superficial lamina propria (original magnification 200). The bile reflux may be visible 20. specifications following safe manufacturing practices. lack of large numbers of neutrophils and plasma cells. Additionally, these changes are dissimilar to those seen in H pylori gastritis, reflecting the mechanistic differences between these gastropathies. BIOPSY: GASTRIC ANTRAL MUCOSA WITH MILD REACTIVE CHANGES; NEGATIVE FOR HELICOBACTER PYLORI TYPE ORGANISMS. [8, 9]. Book An Appointment.no-styles{background-color:transparent !important;}. Never ignore professional medical advice in seeking treatment because of something you have read on the MyPathologyReport site. Maguilnik I, Neumann WL, Sonnenberg A, Genta RM. (adsbygoogle = window.adsbygoogle || []).push({}); (adsbygoogle = window.adsbygoogle || []).push({ If you are not happy, send back all of your bottles within 30 days, of receiving the product and we will issue you a. The microscopic features of reactive gastropathy were well characterized by Dixon et al in their original description of reflux gastritis as a distinct histopathologic entity. . Cytoskeletal and kinetic epithelial differences between NSAID gastropathy and Helicobacter pylori gastritis: an immunohistochemical determination. The gastric mucosa becomes thinner as the normal cells are destroyed. [2] The accompanying histamine-mediated vascular response leads to edema and hyperemia. The objective of this health library is to engage our community and offer relevant, trusted and easy to understand health & wellness information that can be accessed anywhere, anytime, on any device for FREE. Erythemayohus mucosa in the gastric fundus. certain illnesses, such as diabetes or kidney failure. Create an account to follow your favorite communities and start taking part in conversations. Bowel movements: How often should you poop? 2005 Nov. 22(4):273-83. what are my odds of getting colon or gastric cancer. At present, reactive gastropathy is usually encountered in the clinical setting of chronic nonsteroidal anti-inflammatory drug (NSAID) use. This was the only thing on my colonoscopy/endoscopy report that I don't understand. The mucosa may be normal or may exhibit erythema, congestion, edema, or erosions8. Semin Diagn Pathol. What causes antral gastritis and how to treat it? [Full Text]. The duodenogastric reflux results in disruption of the protective mucus barrier and direct injury to the gastric mucosa, causing backflow of hydrogen ions and epithelial damage. To estimate the light action spectrum for the in vivo phototherapy of H. pylori in the visible range, we performed a simulation of the light transmitted by a simple optical model of the gastric wall structure. The following microscopic features are typically seen in reactive gastropathy: With the right information, patients can make the best decisions about their care. This can be done by changing your diet around or changing your lifestyle habits. Differential diagnosis of reactive gastropathy. 1993 Oct. 23(4):341-8. Constant vomiting, nausea, and diarrhea are indications of Foveolar Hyperplasia. Reactive (chemical) gastropathy is a gastric mucosal pattern of injury induced by endogenous (bile / pancreatic reflux) or exogenous (chronic use of NSAIDs, acetylsalicylic acid, ethanol or chemotherapeutics) agents . Gastritis doesn't always cause signs and symptoms. The articles on MyPathologyReport are intended for general informational purposes only and they do not address individual circumstances. Is "gastric antral-type mucosa demonstrating chronic gastritis" a long term autoimmune condition? The reported prevalence of reactive gastropathy among patients taking daily NSAIDs for at least 1 month ranges from 30% to 40% 9. In reactive gastropathy (chemical gastropathy), the distribution of CK8, CK18, and CK19 was normally distributed but increased in intensity. These are lined by mucus cells that are interspersed with endocrine cells (chiefly G and D types), and a few parietal cells. In situations of upper gastrointestinal (GI) stasis, as is seen after gastric surgery, bacterial overgrowth occurs within the proximal small intestine. Abstract Objectives: The gastric mucosal change accompanying gastric antral intestinal metaplasia (IM) in the pediatric population and its clinical implications remain unclear. It consists of simple columnar epithelium, lamina propria, and the . Copyright 2023 Bel Marra Health. The epithelial injury results in excessive exfoliation of the surface epithelial cells, which gives rise to a reactive foveolar hyperplasia17. However, if left untreated, antral gastritis may lead to progressive damage of the stomach lining and potentially create conditions for bacteria to proliferate, resulting in further complication. The most common cause of reactive gastropathy is the movement of bile or pancreatic secretions into the stomach from the small bowel. Nausea. 2013 Aug. 58(8):2266-74. For potential or actual medical emergencies, immediately call 911 or your local emergency service. One may also feel a burning sensation in the upper abdomen. The secondary (deoxycholic and lithocholic) and deconjugated bile acids are more injurious to the gastric mucosa than the primary (colic and chenodeoxycholic) and conjugated bile acids. 27:1348-54. PDF | Sleeve gastrectomy (SG) often induces gastroesophageal reflux, with few and discordant long-term data on the risk of Barrett's esophagus (BE) in. [QxMD MEDLINE Link]. Gastroenterol Res Pract. In situations of upper gastrointestinal (GI) stasis, as is seen after gastric surgery, bacterial overgrowth occurs within the proximal small intestine. what does it mean? It is considered an autoimmune disorder, meaning your bodys immune system attacks healthy cells, causing the symptoms. Educational text answers on HealthTap are not intended for individual diagnosis, treatment or prescription. dad and aunt had precancerous polyps too. Gastric antral vascular ectasia (GAVE) is the underlying cause for 4% of nonvariceal upper GI bleeding. 1979 Apr. Focal intestinal metaplasia usually is caused and occurs in conjunction with chronic gastritis. Rom J Morphol Embryol. The 2023 edition of ICD-10-CM K29.60 became effective on October 1, 2022. What does squamous mucosa mean? 340(24):1888-99. The most common benign cause of mucosal ulcerations is peptic ulcer disease. 2003 Jan. 15(1):35-40. [10]. Sepulveda AR, Patil M. Practical approach to the pathologic diagnosis of gastritis. Antral gastritis refers to the inflammation in the stomach lining found in the antral area of the stomach. [18, 19, 20], At present, reactive gastropathy is usually encountered in the clinical setting of chronic nonsteroidal anti-inflammatory drug (NSAID) use. This increase in intraluminal bacteria leads to subsequent generation of relatively increased concentrations of deconjugated and secondary bile acids within the refluxate. We'll go over some of the most common types of gastropathy, frequent symptoms . 287(6403):1410-2. When you dont get enough B12 to build healthy red blood cells, you risk developing pernicious anemia. AskApollo online Health Information Library is an initiative by Apollo Hospitals for general people, patients, their families and friends who seek useful health information, tips and advice on disease, procedure and certain medications to help you take care of yourself and your loved ones. As mentioned, there are numerous causes of antral gastritis, all having specific treatments associated with them. Chronic gastritis can also be seen in people who have previously been treated for Helicobacter pylori. The updated Sydney System. What Is Intermittent Fasting? All our products are backed by a 100% satisfaction guarantee. Other conditions to consider include the following: The endoscopic findings of reactive gastropathy are mostly nonspecific. Once there, food contents may begin fermenting for extended periods of time, leading to bloating and flatulence. (See Pathophysiology and Etiology. in government-approved facilities. By using our website, you consent to our use of cookies. [15], The epithelial injury results in excessive exfoliation of the surface epithelial cells, which gives rise to a reactive foveolar hyperplasia. 43(4):303-6.